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The division of one cell originally forms the cancerous cells, which participate in repeated metastasis and invasion. The benign tumor, which does not endanger life, grows slowly in one place; yet it takes only one malignant cell to produce a tumor that will consequently invade and migrate to other areas of the body and cause death (see Diagram 13, p.129.)

Table 12. Types of Tumors

Such cancerous cells grow at different speeds, ranging from several weeks to several decades, depending on the type of carcinomatosis involved. Generally, they undergo several stages of change over the period of 10 to 20 years. After 30-fold division, a cancerous cell may attain the weight of one gram; after 40-fold division, it may weigh as much as one kilogram. The span of time between the 30-fold and 40-fold growth rates is the most crucial period for cancer treatment.

After 30-fold division, the resulting tumor is only about the size of a fingertip, and is difficult to detect with the naked eye. However, in just a short time, after 40-fold division it can attain the size of a fist. Little can be done about cancerous tumors at this stage of development. Thus it is important to keep the tumor under control before it reaches this stage.

As mentioned earlier, damaged DNA gives rise to tumors, but how exactly do tumors result from damaged DNA? This can be explained by the human body’s innate tendency to expel toxic substances. The body can distinguish any intruding substance or chemical that is dangerous or toxic, and will consequently excrete this substance to protect itself. If a carcinogenic substance disrupts this toxin protection system of the body and harmful substances accumulate, the DNA in the cells may become damaged. As you know, the DNA is the blueprint or plan for cellular structure and function. Damage to the DNA could result in abnormal cell reproduction and structure. In this manner, normal cells may undergo sudden structural change and generate genes that produce cells of a malignant and invasive nature (see Table 14, p131)

Table 13. Normal cells and cancerous cells




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