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CAUSES OF MALIGNANT TUMORS
The division of one cell originally forms the cancerous cells,
which participate in repeated metastasis and invasion. The benign
tumor, which does not endanger life, grows slowly in one place;
yet it takes only one malignant cell to produce a tumor that will
consequently invade and migrate to other areas of the body and cause
death (see Diagram 13, p.129.)

Such cancerous cells grow at different speeds, ranging from several
weeks to several decades, depending on the type of carcinomatosis
involved. Generally, they undergo several stages of change over
the period of 10 to 20 years. After 30-fold division, a cancerous
cell may attain the weight of one gram; after 40-fold division,
it may weigh as much as one kilogram. The span of time between the
30-fold and 40-fold growth rates is the most crucial period for
cancer treatment.
After 30-fold division, the resulting tumor is only about the size
of a fingertip, and is difficult to detect with the naked eye. However,
in just a short time, after 40-fold division it can attain the size
of a fist. Little can be done about cancerous tumors at this stage
of development. Thus it is important to keep the tumor under control
before it reaches this stage.
As mentioned earlier, damaged DNA gives rise to tumors, but how
exactly do tumors result from damaged DNA? This can be explained
by the human body’s innate tendency to expel toxic substances.
The body can distinguish any intruding substance or chemical that
is dangerous or toxic, and will consequently excrete this substance
to protect itself. If a carcinogenic substance disrupts this toxin
protection system of the body and harmful substances accumulate,
the DNA in the cells may become damaged. As you know, the DNA is
the blueprint or plan for cellular structure and function. Damage
to the DNA could result in abnormal cell reproduction and structure.
In this manner, normal cells may undergo sudden structural change
and generate genes that produce cells of a malignant and invasive
nature (see Table 14, p131)
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